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I'm trying to bulk and I've been stuck at 145 for a month now and I'm trying kick start the gaining porocess again. I've stopped doing cardio, I've upped my caloric intake and my question is should I stop drinking coffee. I have, at the most 4 cups a day, I usually quit around 9 am and switch to water. Does coffee inhibit weight gain?
06-14-2002, 08:33 AM
Caffeine's thermogenic effect is not enough to stop you from gaining weight if you really want to.
06-14-2002, 08:35 AM
It helps mobilize fat. You have to do your part to get rid of it though. Bad part - insulin levels are increased with caffeine.
borrowed from Elzi
Caffeine-induced impairment of glucose tolerance is abolished by
b-adrenergic receptor blockade in humans. Farah S. L. T. and T. E. Graham. J
Appl Physiol 92: 2347-2352, 2002.
Just read the paper the other day and it confirmed my suspicions: that the
transient caffeine-induced decrease in glucose tolerance/clearance is caused
by our old friends: epinephrine and the beta-AR, not adenosine receptor
To make a long story (6 pages) short, male over-night fasted subjects were
given a placebo, 5 mg/kg caffeine, 80 mg of propanolol (a beta-AR blocker),
or same amounts of caffeine and propanolol together. After 90 minutes, an
oral glucose tolerance test was administered (ingestion of 75 g glucose).
Blood samples were taken at various times post-OGTT.
OGTT increased blood glucose in all groups (no difference between
treatments). Caffeine ingestion increased insulin levels by 42% and reduced
whole body insulin sensitivity index by 25%. The major site of net reduction
of glucose was in skeletal muscle. When caffeine was administered with
propanolol (C+P), the increased insulin response was no different than the
placebo and propanolol only. Epinephrine levels were increased in the
caffeine only and C+P groups. Free fatty acids and glycerol levels were
increased as well in caffeine only group (indicative of increased
lipolysis). These changes were absent in C+P.
Mechanisms: increased epinephrine levels and the subsequent inhibition of
insulin-stimulated glucose uptake in skeletal muscle. Another additive
effect may possilby be elevated epi levels enhancing B-cell responsiveness
to glucose and transiently increasing heptatic glucose production, with
insulin response trying to compensate for increased glucose level.
However, note that lipolysis was also increased.
So the balance of the universe is again maintained.
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