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universal
07-10-2003, 02:12 PM
im gonna stack

25 Ephedrine Hcl
5mg Yombine Hcl
200 caffeine

quick ques though...is there any diff bewteen all these caffeinies, iknow some are less then 200 mg but is there any diff bw the ones that are the same dose? same question for the E hcl...


C
http://www.thestimulantpeople.com/cgi-bin/dnestore/scan/se=caffeine/sf=category/sp=dneresults

E

http://www.thestimulantpeople.com/cgi-bin/dnestore/scan/se=ephedrine/sf=category/sp=dneresults

thanks guys

Holto
07-10-2003, 02:19 PM
I have taken several different brands and never noticed a difference

I think quality is pretty relative

bradley
07-10-2003, 06:17 PM
I believe the effective dose of yohimbe is 0.2mg per kg of bw, and yohimbe should not be combined with epedrine due to the effects it can have on heart rate and blood pressure.

From what I understand yohimbe should be taken on an empty stomach, hence the reason that most people take it first thing in the morning before cardio.

universal
07-10-2003, 09:54 PM
not yombie, yombine hcl...i have read articles that say 5mg will help alot...look at san tight for example...5mg per pill

bradley
07-11-2003, 07:02 AM
Originally posted by universal
not yombie, yombine hcl...i have read articles that say 5mg will help alot...look at san tight for example...5mg per pill

I understand but is 5mg enough to get any benefit from the yohimbine?

Yohimbine should not be taken with ephedra or ephedrine products.


Plasma catecholamine levels and lipid mobilization induced by yohimbine in obese and non-obese women.

Berlan M, Galitzky J, Riviere D, Foureau M, Tran MA, Flores R, Louvet JP, Houin G, Lafontan M.

Laboratoire de Pharmacologie Medicale et Clinique, INSERUM U-317, Faculte de Medecine, Toulouse, France.

Oral yohimbine administration (0.2 mg/kg) induced lipid mobilization (increase in plasma non-esterified fatty acids, NEFA) in fasting non-obese women (body mass index BMI = 20.2 +/- 0.5, age 35.5 +/- 2.7 years) without significant action on plasma glucose, insulin levels, heart rate or blood pressure during the time-course of the experiment (240 min). Plasma norpinephrine (but not epinephrine) concentrations were increased (100 percent) after oral yohimbine administration. Oral administration of propranolol (40 mg, 60 min before yohimbine) reduced the lipid-mobilizing action of yohimbine (70 percent) during the 60 min following its administration and then totally suppressed its effect until the end of the experimental period (180 min). In fasting obese women (BMI = 36.4 +/- 2.1, age 37 +/- 3.6 years), yohimbine provoked an increase in plasma NEFA levels which was not markedly different from that observed in non-obese subjects. It had no significant effect on plasma glucose, insulin levels, heart rate or blood pressure. Plasma norepinephrine increased in the same proportions. The lipid-mobilizing effect of yohimbine in women is mainly attributable to the increase in synaptic norepinephrine with a resultant increment in lipolysis by beta-adrenergic agonism. In the standard fasting conditions (12 hours) the blockade of the antilipolytic fat cell alpha 2-adrenoceptors seems to be a minor component of the lipomobilizing effect of yohimbine. Morever, when compared with non-obese women, the lipomobilizing effect of yohimbine is not enhanced in obese women.

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Alpha 2-antagonist compounds and lipid mobilization: evidence for a lipid mobilizing effect of oral yohimbine in healthy male volunteers.

Galitzky J, Taouis M, Berlan M, Riviere D, Garrigues M, Lafontan M.

Laboratoire de Pharmacologie Clinique et Medicale, Universite Paul Sabatier, Toulouse, France.

Investigations were carried out to analyse the interactions of alpha 2-antagonists (yohimbine, idazoxan, SK & F-86,466) with human fat cell alpha 2-adrenoceptors. All the alpha 2-antagonists enhanced the lipolytic potencies of epinephrine with an order of potency: yohimbine greater than idazoxan greater than SK & F-86,466; the same order was also found in 3H-yohimbine competition studies on human fat cell membranes. The most potent agent, yohimbine, was administered orally in humans to define the conditions of appearance and the time-course of a putative lipid-mobilizing action. Oral yohimbine administration (0.2 mg kg-1) elevated plasma glycerol and non-esterified fatty acids in fasting healthy subjects without significant action on heart rate or blood pressure during the time-course of the experiment. The lipid-mobilizing action of yohimbine was reinforced during physical exercise, completely suppressed after a meal and partially blocked by administration of propranolol (0.5 mg kg-1; 60 min before yohimbine). Plasma norepinephrine concentrations were increased (40-50%) after oral yohimbine administration. The rise in plasma catecholamine concentration elicited by yohimbine was not modified by propranolol treatment. The lipid-mobilizing effect of yohimbine could be attributable to: (i) the increase in synaptic norepinephrine with a resultant increment in lipolysis by beta-adrenergic agonism; (ii) a decrease in alpha 2-adrenoceptor stimulation of human fat cell alpha 2-adrenoceptors; (iii) a blockade of presynaptic alpha 2-adrenoceptors. The use of highly selective alpha 2-antagonists will allow investigations into alpha 2-adrenoceptors, which may represent a novel locus for pharmacological intervention in lipid-mobilization strategies.(ABSTRACT TRUNCATED AT 250 WORDS)

universal
07-12-2003, 12:29 AM
thanks for the articles bradley...what im curioous about though is so many people i know take ECY without a second htough....i mean lquid clen(vpx) is prob the most popular fat burner and its a ECY...

im just confused now haha
thnaks though

http://www.vpxsports.com/liquid_clenbutrx.html

bradley
07-12-2003, 06:12 AM
Originally posted by universal
thanks for the articles bradley...what im curioous about though is so many people i know take ECY without a second htough....i mean lquid clen(vpx) is prob the most popular fat burner and its a ECY...


From what I have read most of these products do not contain enough Y to have much of an effect. Y should be taken on an empty stomach to get the beneficial effects.

The supp companies could have another reason for adding in the Y that I am not aware of, but since you are making your own I do not see any reason to add the Y.

You could use the Y first thing in the morning and perform some cardio then ~3 hours later or so start with the EC, which is the way that I have seen people use Y in the past. This is just food for thought so to speak, and I definitely do not claim to the complete ins and outs of Y supplementation.:)

chops
07-19-2003, 11:44 PM
Originally posted by bradley


From what I have read most of these products do not contain enough Y to have much of an effect. Y should be taken on an empty stomach to get the beneficial effects.

so i bought CLENBUTRX tabs before i found this thread and now have questions:

1) what amount of Y is enough to produce an effect?

2) an earlier post stated Y should not be combined with ephedra. since clenbutrx contains ma huang extract , do you recommend against this product? i'm confused if ephedra and mahuang are the *exact* same ingredient.

3) http://www.ephedrafacts.com/consumer1.html says don't exceed 100mg ephedra per day. this products lists 312mg mahuang/3 tabs but i'm unsure if that coverts directly to 312mg of ephedra.


very confused - thanks
(note: i was planning on starting with 2 tabs per day total)

--------------------------------------------------------------------------
CLENBUTRX WILL LITERALLY LIPOSUCK THE FAT RIGHT OUT OF YOUR BODY!

Three Capsules Contain:
Thermo/Neurogenic Blend
MaHuang Extract: 312mg (Standardized at 8%)
Citrus Aurantium Extract: 200mg (Standardized for 10% Synephrine)
L-Tyrosine: 425mg
Guarana (Standardized for 22% Caffeine): 910mg
Bioperine: 5mg
Proprietary Thyro/Anti-estro/Insulinogenic Blend
Guggulsterones (E & Z) (Standardized at 10%)
Coleus Forskohlii
Ipriflavone (7-Isopropoxy Isoflavone)
Yohimbe Extract (Standardized at 8%)
Bis-Glycinato Oxovanadium
Alpha Lipoic Acid
Iodine (Kelp)
---------------------------------------------------------------

Goin_Big
07-20-2003, 12:21 AM
hehehe, 312mb of ephedrine that would hurt :D

standardized for 8% means that you are getting 25mg of ephedrine.

I dunno much about yohimbe/yohimbine.

I'm currently taking and ECT T = tyrosine.

Ma Huang is pretty much ephedrine and will interact the same with yohimbe.

bradley
07-20-2003, 12:32 PM
Originally posted by chops
1) what amount of Y is enough to produce an effect?

~0.2mg per kg of bw


2) an earlier post stated Y should not be combined with ephedra. since clenbutrx contains ma huang extract , do you recommend against this product? i'm confused if ephedra and mahuang are the *exact* same ingredient.


Ma huang is an herb and the primary active ingredient is ephedra.

I doubt the products that contain yohimbe and ephedra contain enough yohimbe to be effective, or cause any adverse effects. Although the supplement manufacturers may have some other reason for adding the Y that I am unaware of. I have never used Clenbutrx myself, but the comments I have seen on it's effectiveness have been postive.